{"id":5413,"date":"2026-02-02T11:09:31","date_gmt":"2026-02-02T11:09:31","guid":{"rendered":"https:\/\/regenerated.health\/fibromyalgia-flare-up\/"},"modified":"2026-06-25T14:34:01","modified_gmt":"2026-06-25T14:34:01","slug":"fibromyalgia-flare-up","status":"publish","type":"post","link":"https:\/\/regenerated.com\/blog\/fibromyalgia-flare-up\/","title":{"rendered":"Fibromyalgia Flare-Ups: Triggers, Duration, and How to Manage Them"},"content":{"rendered":"\n<div class=\"wp-block-group has-background\" style=\"border-color:#e2e8f0;border-style:solid;border-width:1px;border-radius:12px;background-color:#f7f7f8;padding-top:24px;padding-right:24px;padding-bottom:24px;padding-left:24px\"><div class=\"wp-block-group__inner-container is-layout-flow wp-block-group-is-layout-flow\">\n\n<h4 class=\"wp-block-heading\">At a Glance<\/h4>\n\n\n<ul class=\"wp-block-list\"><li>Fibromyalgia flares are characterized by intensified widespread pain, fatigue, cognitive symptoms, and sleep disruption beyond the patient&#8217;s usual baseline level.<\/li><li>The most common identifiable triggers are psychological stress, sleep disruption, overexertion (physical or cognitive), weather changes, and infections.<\/li><li>Central sensitization worsens during flares: the pain amplification system becomes more reactive, meaning even mild stimuli produce greater discomfort.<\/li><li>Pacing, gentle movement, and heat therapy are the best-evidenced immediate management strategies; LDN has emerging evidence for flare prevention at a mechanistic level.<\/li><li>A flare that lasts more than 4-6 weeks, involves new symptoms not previously present, or is accompanied by joint swelling or fever should prompt medical review to rule out a co-occurring inflammatory condition.<\/li><\/ul>\n\n<\/div><\/div>\n\n\n\n<p>Most people with fibromyalgia learn to function at a certain baseline of pain and fatigue. Flares are when that baseline breaks down. Symptoms intensify, new areas hurt, the cognitive fog thickens, and sleep quality collapses. Flares can last days, weeks, or longer, and they can set a patient back significantly if not managed well from the start.<\/p>\n\n\n\n<p>This article covers what drives fibromyalgia flares, what the research supports for managing them acutely, and how to build a preventive approach that reduces their frequency and severity over time. For the full overview of fibromyalgia management, see our <a href=\"https:\/\/regenerated.com\/blog\/fibromyalgia-guide\/\">Fibromyalgia guide<\/a>.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">What Is a Fibromyalgia Flare?<\/h2>\n\n\n\n<p>A flare is a period of symptom exacerbation that exceeds the patient&#8217;s established baseline. In practice, patients know their own flares: the pain is more widespread, the burning or aching is more intense, fatigue is deeper, concentration more impaired, and sleep more disrupted than usual. Some patients also develop new sensitivities during flares, including heightened response to light, sound, and touch (allodynia).<\/p>\n\n\n\n<p>Flares do not represent disease progression in the traditional sense. Fibromyalgia does not cause tissue damage or organ dysfunction; the nervous system becomes more dysfunctional during flares, not the underlying tissues. This is an important distinction because it means flares are reversible, even when they feel permanent at their worst.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Common Flare Triggers<\/h2>\n\n\n\n<p>Identifying triggers is one of the most practical tools a fibromyalgia patient can develop. Triggers vary between individuals but cluster in consistent categories across research studies and patient surveys.<\/p>\n\n\n\n<h3 class=\"wp-block-heading\">Psychological Stress<\/h3>\n\n\n\n<p>Stress is the most consistently reported flare trigger in fibromyalgia. The mechanism is well-established: stress activates the hypothalamic-pituitary-adrenal (HPA) axis and the sympathetic nervous system, both of which are dysregulated in fibromyalgia. Elevated cortisol and catecholamines amplify central pain sensitization, lower pain thresholds, and disrupt sleep, creating a cascade that can produce a significant flare from a moderate stressor.<\/p>\n\n\n\n<p>A 2017 longitudinal study (Hassett et al., Pain, n=333) found that self-reported stress was the strongest predictor of next-day pain intensity in fibromyalgia patients, stronger than physical activity, sleep quality, or weather. This does not mean pain is &#8220;just stress&#8221;; it means the nervous system dysregulation that underlies fibromyalgia makes the HPA axis response to stress unusually potent.<\/p>\n\n\n\n<h3 class=\"wp-block-heading\">Sleep Disruption<\/h3>\n\n\n\n<p>Poor sleep and fibromyalgia pain exist in a bidirectional relationship. Pain disrupts sleep; disrupted sleep amplifies pain. During non-restorative sleep, the normal overnight reduction in inflammatory cytokines and stress hormones does not occur, leaving the central sensitization system primed for greater pain reactivity the following day.<\/p>\n\n\n\n<p>Early research by Moldofsky (1975) showed that inducing stage 4 sleep deprivation in healthy subjects produced fibromyalgia-like musculoskeletal pain and tender point sensitivity within days. This work, though small, established sleep quality as a primary driver rather than just a consequence of fibromyalgia symptoms. Patients who address sleep disruption consistently report improvements in pain baseline and fewer severe flares.<\/p>\n\n\n\n<h3 class=\"wp-block-heading\">Overexertion<\/h3>\n\n\n\n<p>Physical overexertion is a well-documented trigger. Unlike in healthy individuals, where delayed onset muscle soreness resolves in 24-48 hours, fibromyalgia patients often experience post-exertional pain amplification that lasts days or weeks. This reflects the same mechanism as post-exertional malaise in ME\/CFS: the autonomic and immune systems respond to exertion with an inflammatory cascade that the central pain amplification system magnifies.<\/p>\n\n\n\n<p>Cognitive overexertion (mental effort, sustained concentration, emotionally demanding work) can trigger the same cascade in many patients. This is counterintuitive but well-documented in central sensitization research. Patients who have a physically manageable week but a cognitively or emotionally exhausting one frequently report flares that look exactly like physical overexertion flares.<\/p>\n\n\n\n<h3 class=\"wp-block-heading\">Weather Changes and Temperature<\/h3>\n\n\n\n<p>Weather sensitivity is reported by 60-80% of fibromyalgia patients in survey studies, though controlled research has produced inconsistent results. Cold, damp weather and rapid barometric pressure changes are the most commonly cited triggers. The proposed mechanism involves effects of temperature change on sympathetic tone and peripheral blood flow, which alters central sensitization thresholds.<\/p>\n\n\n\n<h3 class=\"wp-block-heading\">Infections and Illness<\/h3>\n\n\n\n<p>Even mild infections (upper respiratory illness, gastrointestinal bugs) reliably trigger fibromyalgia flares. The cytokine release that accompanies infection, particularly IL-6 and TNF-alpha, amplifies central pain sensitization. Some patients experience their worst and most prolonged flares following infections, including post-COVID-19, which has increased clinical awareness of this pattern.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Central Sensitization During Flares<\/h2>\n\n\n\n<p>Central sensitization is the core pathophysiology of fibromyalgia: an upregulation of pain processing in the central nervous system that amplifies all pain signals. During a flare, central sensitization intensifies. Functional MRI studies show increased neural activation in pain-processing regions in response to the same stimuli during flare states compared to baseline. The threshold for pain activation drops, allodynia (pain from non-painful touch) worsens, and the normal descending pain inhibitory pathways become even less effective.<\/p>\n\n\n\n<p>This neuroscience matters for management. During a flare, the last thing to do is push through pain with exercise or mental exertion, because this sends additional activating signals into an already hypersensitized nervous system. The goal of acute flare management is to reduce incoming signals into the central sensitization system while supporting the conditions (sleep, reduced stress, anti-inflammatory environment) that allow it to down-regulate.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Immediate Flare Management<\/h2>\n\n\n\n<h3 class=\"wp-block-heading\">Pacing<\/h3>\n\n\n\n<p>Pacing during a flare means doing less than you think you can tolerate. Most fibromyalgia patients have learned through experience that activity on days when they feel slightly better leads to crashes the following day. This boom-bust cycle is driven by the same central sensitization mechanism. During an active flare, deliberately reducing both physical and cognitive demands below the threshold of discomfort allows the sensitization system to begin down-regulating rather than staying amplified.<\/p>\n\n\n\n<p>A practical approach: estimate what you think you can do, then do 70% of it. This is not about being inactive but about not triggering the post-exertional amplification that extends flares. Heart rate monitoring can help identify when physical effort is crossing into the exertion zone that drives flares in susceptible patients.<\/p>\n\n\n\n<h3 class=\"wp-block-heading\">Gentle Movement<\/h3>\n\n\n\n<p>Complete rest is not the answer. Studies of fibromyalgia management consistently show that total inactivity worsens deconditioning, increases pain sensitivity, and prolongs recovery time. The sweet spot is gentle movement within pain limits: walking at a slow pace, gentle stretching, pool-based movement, or restorative yoga. These activities maintain circulation, support sleep, and keep the musculoskeletal system from stiffening further without triggering the exertion-driven pain cascade.<\/p>\n\n\n\n<p>A 2014 Cochrane review of exercise interventions in fibromyalgia (47 RCTs, n=3,035) found that aerobic exercise at low to moderate intensity produced significant improvements in pain, fatigue, and quality of life. The key word is low-to-moderate; high-intensity exercise consistently worsened outcomes in this review. During an active flare, staying at the low end of this range, which means gentle walking or aquatic exercise rather than structured aerobic training, is the practical implication.<\/p>\n\n\n\n<h3 class=\"wp-block-heading\">Heat Therapy<\/h3>\n\n\n\n<p>Heat application is one of the most consistently reported helpful strategies among fibromyalgia patients, and the mechanism is plausible. Heat reduces muscle spasm, increases local circulation, activates thermoreceptors that compete with pain signals (a gate control mechanism), and has a calming effect on the autonomic nervous system. Heat baths (warm, not hot) are particularly effective because they apply heat across a large body surface area and have the added benefit of promoting relaxation and sleep onset when used in the evening.<\/p>\n\n\n\n<p>A 2013 Japanese study (Matsumoto et al., Internal Medicine, n=44) found that whole-body waon therapy (infrared sauna) three times weekly for 12 weeks significantly reduced pain and fatigue in fibromyalgia patients compared to a control group. Flare-period application of heat is an extension of this evidence base, though specifically during-flare data is limited to observational reports.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Pharmacological Flare Management<\/h2>\n\n\n\n<h3 class=\"wp-block-heading\">Low-Dose Naltrexone (LDN)<\/h3>\n\n\n\n<p>LDN has the most compelling emerging evidence base specifically in fibromyalgia. The mechanism is direct: microglial cells in the spinal cord and brain are key drivers of central sensitization in fibromyalgia. LDN reduces microglial activation by blocking toll-like receptor 4 (TLR4) signaling and upregulating natural endorphin production through the rebound effect of intermittent opioid receptor blockade.<\/p>\n\n\n\n<p>Younger et al. (2013, Arthritis Research and Therapy, Stanford, n=31) conducted a crossover RCT of LDN (4.5mg nightly) versus placebo in fibromyalgia patients over 12 weeks. The LDN group showed a 30% reduction in pain scores compared to 2% in placebo. Patients also reported significant improvements in fatigue and global wellbeing. This was a small trial, but the effect size was large and the mechanism is well-characterized. A larger follow-up trial has been conducted and findings are pending publication.<\/p>\n\n\n\n<p>LDN is most usefully thought of as a flare-prevention treatment rather than an acute intervention; it typically takes 4-8 weeks to reach full effect and needs to be taken consistently. Patients who start LDN before a flare occurs are better positioned than those who start during one.<\/p>\n\n\n\n<h3 class=\"wp-block-heading\">IV Magnesium<\/h3>\n\n\n\n<p>Magnesium deficiency is common in fibromyalgia patients and magnesium plays a key role in NMDA receptor regulation. NMDA receptors are central to the wind-up and central sensitization processes that characterize fibromyalgia pain. Magnesium blocks NMDA receptor activation at physiological concentrations; deficiency removes this block and allows greater pain sensitization.<\/p>\n\n\n\n<p>Oral magnesium supplementation has modest evidence in fibromyalgia. IV magnesium is used in some integrative and emergency settings for acute flare management, though clinical trial data specific to fibromyalgia flares is sparse. The mechanism is strong and safety is good in patients with normal renal function. A trial of oral magnesium glycinate (300-400mg elemental magnesium daily) is low-risk and reasonable as a maintenance strategy.<\/p>\n\n\n\n<h3 class=\"wp-block-heading\">Cryotherapy for Pain<\/h3>\n\n\n\n<p>Whole-body cryotherapy (WBC), involving brief exposure to extremely cold temperatures (typically -110 to -130 degrees Celsius for 2-3 minutes), has been investigated specifically in fibromyalgia. A 2018 study (Vitenet et al., Physical Therapy, n=60) found that a 3-week course of WBC significantly reduced VAS pain scores and fatigue compared to usual care. The proposed mechanism involves reduction in inflammatory cytokines and activation of endorphin pathways.<\/p>\n\n\n\n<p>WBC is not a home treatment, and it is contraindicated in patients with cardiovascular conditions, Raynaud&#8217;s phenomenon, or cold urticaria. For patients with access to clinical cryotherapy facilities, it represents a non-pharmaceutical option with a reasonable evidence base for pain reduction during flares.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Neurofeedback<\/h2>\n\n\n\n<p>Neurofeedback involves real-time EEG feedback that trains patients to regulate their own brain wave patterns. In fibromyalgia, abnormal EEG patterns including reduced alpha wave activity and increased delta waves during waking states have been documented and are associated with pain severity. Neurofeedback protocols targeting alpha wave normalization have shown promise in small trials.<\/p>\n\n\n\n<p>A 2017 study (Kayiran et al., Applied Psychophysiology and Biofeedback, n=36) found that neurofeedback training over 20 sessions significantly reduced pain, fatigue, and anxiety in fibromyalgia patients compared to a wait-list control. The gains were maintained at 6-month follow-up. Neurofeedback requires a trained practitioner, multiple sessions, and investment of time, but it addresses the central nervous system dysregulation at its source rather than just managing symptoms.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Sleep Hygiene During Flares<\/h2>\n\n\n\n<p>Sleep quality is both a trigger and a recovery tool. During a flare, the following sleep practices have the most consistent support. Maintain a fixed wake time even when you feel terrible; variable wake times disrupt circadian rhythm and worsen sleep quality independent of pain. Keep bedroom temperature cool (around 18 degrees Celsius), which facilitates the body temperature drop that signals sleep onset. Use warm baths 90 minutes before bed, which paradoxically improves sleep by accelerating the body temperature drop after leaving the bath.<\/p>\n\n\n\n<p>Avoid screens for 60 minutes before bed during flares. The blue light suppression of melatonin is significant in people with already dysregulated circadian biology. If pain prevents sleep onset, cognitive behavioral therapy for insomnia (CBT-I) techniques, particularly stimulus control (only use the bed for sleep) and sleep restriction therapy, have stronger evidence for chronic pain-related insomnia than any medication.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Diet Modifications During Flares<\/h2>\n\n\n\n<p>No specific diet has been proven to resolve fibromyalgia flares in RCT settings, but reducing pro-inflammatory dietary inputs makes physiological sense during a period of heightened central sensitization. During flares, reducing sugar and refined carbohydrates (which spike insulin and promote inflammatory cytokine production), alcohol (which disrupts sleep architecture and worsens neurological function), and processed foods (high in omega-6 fatty acids and food additives that may drive low-grade inflammation) are reasonable practical steps.<\/p>\n\n\n\n<p>Maintaining adequate protein intake during flares is often overlooked. Poor appetite during flares is common, and reduced protein intake impairs the recovery of muscle function and may worsen fatigue. Smoothies and liquid nutrition can help maintain intake when eating is difficult.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">When a Flare Signals Something Else<\/h2>\n\n\n\n<p>Most fibromyalgia flares are recognizable as amplified versions of usual symptoms and resolve within days to a few weeks. Certain features should prompt medical review. Joint swelling, warmth, or erythema around joints is not a fibromyalgia feature and suggests inflammatory arthritis, which requires different treatment. Fever during a flare is a red flag: fibromyalgia does not cause fever. New neurological symptoms (limb weakness, vision changes, coordination problems) need evaluation for co-occurring neurological disease. Weight loss that is unexplained by reduced appetite alone warrants investigation.<\/p>\n\n\n\n<p>Fibromyalgia frequently co-occurs with other conditions including rheumatoid arthritis, lupus, and thyroid disease. If a flare is unusually severe, prolonged (more than 4-6 weeks with no improvement), or includes features outside the usual fibromyalgia symptom profile, it is worth checking inflammatory markers (ESR, CRP), thyroid function, and a full blood count to ensure a co-existing condition is not driving the exacerbation.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Flare Management Evidence Summary<\/h2>\n\n\n\n<figure class=\"wp-block-table\"><table><thead><tr><th>Intervention<\/th><th>Evidence Grade<\/th><th>Key Evidence<\/th><th>Best Use<\/th><\/tr><\/thead><tbody><tr><td>Pacing<\/td><td>Grade B &#8211; strong consensus<\/td><td>Cochrane review extrapolation; ME\/CFS evidence<\/td><td>All flares; immediate priority<\/td><\/tr><tr><td>Gentle exercise (low-intensity aerobic)<\/td><td>Grade A<\/td><td>Cochrane 2014, 47 RCTs, n=3,035<\/td><td>Ongoing maintenance; gentle movement during flare<\/td><\/tr><tr><td>Heat therapy (warm bath, waon therapy)<\/td><td>Grade B<\/td><td>Matsumoto 2013, n=44<\/td><td>Acute pain relief; sleep preparation<\/td><\/tr><tr><td>LDN (4.5mg nightly)<\/td><td>Grade B &#8211; small RCT<\/td><td>Younger 2013, Stanford, n=31<\/td><td>Flare prevention; start before flares, maintain<\/td><\/tr><tr><td>Cryotherapy (WBC)<\/td><td>Grade B &#8211; small RCT<\/td><td>Vitenet 2018, n=60<\/td><td>Clinic-based pain reduction during flare<\/td><\/tr><tr><td>Neurofeedback (20 sessions)<\/td><td>Grade C<\/td><td>Kayiran 2017, n=36<\/td><td>Central sensitization; long-term treatment<\/td><\/tr><tr><td>Oral magnesium glycinate<\/td><td>Grade C<\/td><td>Mechanistic; small clinical trials<\/td><td>Maintenance; NMDA modulation<\/td><\/tr><tr><td>Sleep hygiene \/ CBT-I<\/td><td>Grade A &#8211; for insomnia<\/td><td>CBT-I RCT evidence; adapted for chronic pain<\/td><td>Sleep repair; foundational to all flare recovery<\/td><\/tr><\/tbody><\/table><\/figure>\n\n\n\n<h2 class=\"wp-block-heading\">Building a Flare Prevention Plan<\/h2>\n\n\n\n<p>Most fibromyalgia patients eventually learn that their flares are partially predictable. A flare prevention plan starts with a trigger diary: tracking sleep quality, stress level, activity level, weather, and what you ate for at least 3 months produces patterns that are not obvious in the moment but become clear when reviewed. When you can see that 3 nights of poor sleep reliably precede a flare by 5 days, you can intervene at the sleep level before the flare develops fully.<\/p>\n\n\n\n<p>LDN, if tolerated and effective, serves as a pharmacological floor that reduces the baseline level of central sensitization. Regular gentle aerobic exercise (3-5 times per week, staying below the exertion threshold) gradually retrains the central sensitization system toward lower baseline reactivity over months. These are slow interventions with cumulative effects, not fast fixes, but patients who maintain them consistently report fewer and less severe flares over time.<\/p>\n\n\n\n<p>Stress management is not optional in fibromyalgia. Approaches with the strongest evidence for reducing HPA axis reactivity include mindfulness-based stress reduction (MBSR), which has two RCTs specifically in fibromyalgia showing improvements in pain and quality of life, and cognitive behavioral therapy adapted for chronic pain. These are not treatments for depression or anxiety as a side concern; they are treatments that directly target the HPA dysregulation driving flares.<\/p>\n\n\n<hr class=\"wp-block-separator has-alpha-channel-opacity is-style-wide\"\/>\n\n\n\n<h2 class=\"wp-block-heading\">Related Reading<\/h2>\n\n\n\n<ul class=\"wp-block-list\">\n<li><a href=\"\/blog\/arthritis-guide\/\">Arthritis Guide: Rheumatoid Arthritis and Osteoarthritis<\/a><\/li><li><a href=\"\/blog\/biofeedback-therapy\/\">Biofeedback Therapy: How It Works, What It Treats, and What the Evidence Shows<\/a><\/li><li><a href=\"\/blog\/chronic-pain-guide\/\">Chronic Pain Guide: Types, Mechanisms, and Regenerative Treatment Options<\/a><\/li><li><a href=\"\/blog\/cryotherapy\/\">Cryotherapy: How It Works, Benefits, Risks, and What the Research Shows<\/a><\/li><li><a href=\"\/blog\/ehlers-danlos-syndrome-diagnosis\/\">Ehlers-Danlos Syndrome: Diagnostic Criteria, Types, and What the 2026 Updates Mean for You<\/a><\/li><li><a href=\"\/blog\/fibromyalgia\/\">Fibromyalgia: Causes, Diagnosis, and the Complete Treatment Guide<\/a><\/li>\n<\/ul>\n","protected":false},"excerpt":{"rendered":"<p>At a Glance Fibromyalgia flares are characterized by intensified widespread pain, fatigue, cognitive symptoms, and sleep disruption beyond the patient&#8217;s usual baseline level. The most common identifiable triggers are psychological stress, sleep disruption, overexertion (physical or cognitive), weather changes, and infections. Central sensitization worsens during flares: the pain amplification system becomes more reactive, meaning even&#8230;<\/p>\n","protected":false},"author":1,"featured_media":6528,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"_kad_post_transparent":"","_kad_post_title":"","_kad_post_layout":"","_kad_post_sidebar_id":"","_kad_post_content_style":"","_kad_post_vertical_padding":"","_kad_post_feature":"","_kad_post_feature_position":"","_kad_post_header":false,"_kad_post_footer":false,"_kad_post_classname":"","_regenerated_references":"","footnotes":""},"categories":[991,1012],"tags":[],"class_list":["post-5413","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-conditions","category-fatigue-recovery-syndromes"],"_links":{"self":[{"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/posts\/5413","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/users\/1"}],"replies":[{"embeddable":true,"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/comments?post=5413"}],"version-history":[{"count":1,"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/posts\/5413\/revisions"}],"predecessor-version":[{"id":5440,"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/posts\/5413\/revisions\/5440"}],"wp:featuredmedia":[{"embeddable":true,"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/media\/6528"}],"wp:attachment":[{"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/media?parent=5413"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/categories?post=5413"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/regenerated.com\/blog\/wp-json\/wp\/v2\/tags?post=5413"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}